Doctor Profile

Joseph B. Davis

Joseph Davis

Reproductive Medicine Associates of New York - Downtown
594 Broadway, Suite 1011
New York, NY 10012

New York

New York (NY)

Contact Contact Joseph B. Davis
T: 212-906-7900
F: 212-965-1800

Reproductive Endocrinology; Obstetrics/Gynecology

Reproductive Endocrinology



Dr. Joseph Davis is a board certified Reproductive Endocrinologist and infertility specialist with a passion for helping couples and individuals to achieve their goals of becoming a family. Dr. Davis works with patients to addressing their medical, psychological, nutritional, and social needs as part of a comprehensive approach to care. He specializes in medical as well as surgical care for fertility including the Da Vinci robotic surgical system for the treatment of fibroids, endometriosis, and tubal damage.

Dr. Davis obtained is Doctorate of Osteopathic Medicine from Ohio University and his fellowship training in Reproductive Endocrinology and Infertility at Albert Einstein College of Medicine. He has since received numerous awards including the laparoscopic surgeon award from the American Association of Gynecologic Laparoscopists, the New England Fertility Society/Ferring research grant, the Endocrine Society clinical research in Women’s Health award, as well as the outstanding medical student teacher award for 2013-14. As an Assistant Professor in the Division of Reproductive Endocrinology and Infertility Dr. Davis was granted a prestigious global health internship in Geneva, Switzerland, where he became a member of the Guideline Development Group for the World Health Organization and worked in the field of human rights and ethics for infertility.

Dr. Davis is board certified in Obstetrics and Gynecology and Reproductive Endocrinology and is a fellow of the American College of Obstetricians and Gynecologists. He is also a member the American Society for Reproductive Medicine where he serves on the national Ethics committee, the Society of Reproductive Endocrinology and Infertility as well as the European Society of Human Reproduction and Embryology. Dr. Davis has been a peer reviewer for the Journal of Assisted Reproduction and Genetics and is an accomplished lecturer and author having written numerous scientific research articles and manuscripts in leading medical journals. He has presented many of his research findings at national meetings, including the American Society for Reproductive Medicine (ASRM). 

Published In:

New York Rising Stars 2018
New York Rising Stars 2017

About Joseph Davis

Medical School: Ohio University, College of Osteopathic Medicine

Graduation Year: 2006

Professional Webpage:

Honors and Awards:

Awarded this grant for my Mentor based on my fellowship thesis

Clinical Research Fellowship and Mentor Award in Women’s Health, Pfizer/Endocrine Society, 2013

Awarded this in residency after displaying exceptional skill with minimally invasive surgery. 

Laparoscopic Surgeon Award, American Association of Gynecologic Laparoscopists, 2010

Special Licenses/Certifications:

Certificate of da Vinci System Training As a Console Surgeon


Professional Activity:

Selected as a member of the American Society of Reproductive Medicine Ethics Committee


Map Location

Additional Locations

Reproductive Medicine Associates of New York
635 Madison Ave.
10th Floor
New York, NY 10022

White Papers

Maternal Vitamin D Deficiency Programs Reproductive Dysfunction in Female Mice Offspring Through Adverse Effects on the Neuroendocrine Axis. (2016)

Vitamin D (VitD) deficiency affects more than 1 billion people worldwide with a higher prevalence in reproductive-aged women and children. The physiological effects of maternal VitD deficiency on the reproductive health of the offspring has not been studied. In this study we examined maternal VitD deficiency at three specific developmental stages: 1) in utero, 2) preweaning, or 3) in utero and preweaning. VitD deficiency did not affect puberty however all female mice exposed to maternal VitD deficiency developed prolonged and irregular estrous cycles characterized by oligoovulation and extended periods of diestrus. These findings suggest that maternal VitD deficiency programs reproductive dysfunction in adult female offspring through adverse effects on hypothalamic function.   

Intersecting gender, evaluations, and examinations: Averting gender bias in an obstetrics and gynecology clerkship in the United States. (2016)

The purpose of this study was to determine whether gender bias was present in the final third-year medical student obstetrics/gynecology clerkship performance evaluation completed by faculty and resident physicians. This was a retrospective cohort study at a private medical school in Wisconsin. Each student's performance during their required 6-week obstetrics/gynecology clerkship was assessed. Final obstetrics/gynecology clerkship average clinical evaluation scores and obstetrics/gynecology standardized national subject examination scores were compared to see if a gender based difference between subject examination and performance evaluation scores existed. Medical student performance on the standardized national subject exam correlated with clinical evaluations independent of gender. Women had higher scores on both the subject examination and the clinical performance evaluations. There was no evidence of gender bias in the students' clinical evaluation scores.

Disclosure of experience with oocyte cryopreservation. (2014)

Ethical arguments relating to the duty to disclose experience with oocyte cryopreservation. 

Peripubertal vitamin D3 deficiency delays puberty and disrupts the estrous cycle in adult female mice (2012)

We tested the hypothesis that peripubertal vitamin D(3) deficiency disrupts hypothalamic-pituitary-ovarian physiology. We assessed the age of vaginal opening and first estrus (puberty markers), gonadotropin levels, ovarian histology, ovarian responsiveness to exogenous gonadotropins, and estrous cyclicity in mice born with vitamin D(3) deficiency. Peripubertal vitamin D(3) deficiency significantly delayed vaginal opening without affecting the number of GnRH-immunopositive neurons or estradiol-negative feedback on gonadotropin levels during diestrus. Young adult females maintained on a vitamin D(3)-deficient diet after puberty had arrested follicular development and prolonged estrous cycles characterized by extended periods of diestrus. Estrous cycles were restored when vitamin D(3)-deficient young adult females were transferred to a vitamin D(3)-replete diet. This study is the first to demonstrate that peripubertal vitamin D(3) sufficiency is important for an appropriately timed pubertal transition and maintenance of normal female reproductive physiology. These data suggest vitamin D(3) is a key regulator of neuroendocrine and ovarian physiology.

Novel, orally active selective progesterone receptor modulator CP8947 inhibits leiomyoma cell proliferation without adversely affecting endometrium or myometrium. (2010)

Uterine leiomyomas are highly prevalent and often symptomatic, but current medical therapies are limited. A novel, potent, selective, orally active therapy is needed. The goal of these studies was to determine the benefit of the novel non-steroidal selective progesterone receptor modulators (SPRMs) CP8863 and CP8947. CP8947 was a potent novel non-steroidal SPRM that was selective for PR, demonstrated progestational activity in endometrium, inhibited leiomyoma cell proliferation and decreased ECM component production, without disrupting myometrial cell proliferation.

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